![Neuroinflammatory mechanisms of blood-brain barrier damage in ischemic stroke | American Journal of Physiology-Cell Physiology Neuroinflammatory mechanisms of blood-brain barrier damage in ischemic stroke | American Journal of Physiology-Cell Physiology](https://journals.physiology.org/cms/10.1152/ajpcell.00136.2018/asset/images/medium/zh00121883540001.gif)
Neuroinflammatory mechanisms of blood-brain barrier damage in ischemic stroke | American Journal of Physiology-Cell Physiology
![Mfsd2a and Spns2 are essential for sphingosine-1-phosphate transport in the formation and maintenance of the blood-brain barrier Mfsd2a and Spns2 are essential for sphingosine-1-phosphate transport in the formation and maintenance of the blood-brain barrier](https://www.science.org/cms/10.1126/sciadv.aay8627/asset/77f7cac7-b354-4b7c-800e-f0e5f88bb44b/assets/graphic/aay8627-f1.jpeg)
Mfsd2a and Spns2 are essential for sphingosine-1-phosphate transport in the formation and maintenance of the blood-brain barrier
![Platelet regulates neuroinflammation and restores blood–brain barrier integrity in a mouse model of traumatic brain injury - Gao - 2020 - Journal of Neurochemistry - Wiley Online Library Platelet regulates neuroinflammation and restores blood–brain barrier integrity in a mouse model of traumatic brain injury - Gao - 2020 - Journal of Neurochemistry - Wiley Online Library](https://onlinelibrary.wiley.com/cms/asset/82b635a8-a4df-4938-8395-91d95cb51917/jnc14983-fig-0007-m.jpg)
Platelet regulates neuroinflammation and restores blood–brain barrier integrity in a mouse model of traumatic brain injury - Gao - 2020 - Journal of Neurochemistry - Wiley Online Library
![Neural Progenitor Cell-Derived Extracellular Vesicles Enhance Blood-Brain Barrier Integrity by NF-κB (Nuclear Factor-κB)-Dependent Regulation of ABCB1 (ATP-Binding Cassette Transporter B1) in Stroke Mice | Arteriosclerosis, Thrombosis, and Vascular Biology Neural Progenitor Cell-Derived Extracellular Vesicles Enhance Blood-Brain Barrier Integrity by NF-κB (Nuclear Factor-κB)-Dependent Regulation of ABCB1 (ATP-Binding Cassette Transporter B1) in Stroke Mice | Arteriosclerosis, Thrombosis, and Vascular Biology](https://www.ahajournals.org/cms/asset/16b6b3b3-ecd8-4671-b3bd-85b0db0ff5b8/atvbaha.120.315031.fig08.jpg)
Neural Progenitor Cell-Derived Extracellular Vesicles Enhance Blood-Brain Barrier Integrity by NF-κB (Nuclear Factor-κB)-Dependent Regulation of ABCB1 (ATP-Binding Cassette Transporter B1) in Stroke Mice | Arteriosclerosis, Thrombosis, and Vascular Biology
![Fenretinide attenuates lipopolysaccharide (LPS)-induced blood-brain barrier (BBB) and depressive-like behavior in mice by targeting Nrf-2 signaling - ScienceDirect Fenretinide attenuates lipopolysaccharide (LPS)-induced blood-brain barrier (BBB) and depressive-like behavior in mice by targeting Nrf-2 signaling - ScienceDirect](https://ars.els-cdn.com/content/image/1-s2.0-S0753332219353028-ga1.jpg)
Fenretinide attenuates lipopolysaccharide (LPS)-induced blood-brain barrier (BBB) and depressive-like behavior in mice by targeting Nrf-2 signaling - ScienceDirect
![Attenuation of Blood-Brain Barrier Breakdown and Hyperpermeability by Calpain Inhibition* - Journal of Biological Chemistry Attenuation of Blood-Brain Barrier Breakdown and Hyperpermeability by Calpain Inhibition* - Journal of Biological Chemistry](https://els-jbs-prod-cdn.jbs.elsevierhealth.com/cms/attachment/f58601bc-0089-4282-b197-03efbc621ecd/gr1_lrg.jpg)
Attenuation of Blood-Brain Barrier Breakdown and Hyperpermeability by Calpain Inhibition* - Journal of Biological Chemistry
Assessment of Blood-Brain Barrier Function and the Neuroinflammatory Response in the Rat Brain by Using Cerebral Open Flow Microperfusion (cOFM)
![Effects of lipopolysaccharide on the expression of plasma membrane monoamine transporter (PMAT) at the blood–brain barrier and its implications to the transport of neurotoxins - Wu - 2015 - Journal of Neurochemistry - Effects of lipopolysaccharide on the expression of plasma membrane monoamine transporter (PMAT) at the blood–brain barrier and its implications to the transport of neurotoxins - Wu - 2015 - Journal of Neurochemistry -](https://onlinelibrary.wiley.com/cms/asset/fd1f28ef-89d2-4221-a55b-8a54dbf4d46a/jnc13363-fig-0006-m.jpg)
Effects of lipopolysaccharide on the expression of plasma membrane monoamine transporter (PMAT) at the blood–brain barrier and its implications to the transport of neurotoxins - Wu - 2015 - Journal of Neurochemistry -
![Frontiers | Impact of Metabolic Syndrome on Neuroinflammation and the Blood–Brain Barrier | Neuroscience Frontiers | Impact of Metabolic Syndrome on Neuroinflammation and the Blood–Brain Barrier | Neuroscience](https://www.frontiersin.org/files/Articles/423804/fnins-12-00930-HTML/image_m/fnins-12-00930-g001.jpg)
Frontiers | Impact of Metabolic Syndrome on Neuroinflammation and the Blood–Brain Barrier | Neuroscience
![Blood–brain barrier disruption in atrial fibrillation: a potential contributor to the increased risk of dementia and worsening of stroke outcomes? | Open Biology Blood–brain barrier disruption in atrial fibrillation: a potential contributor to the increased risk of dementia and worsening of stroke outcomes? | Open Biology](https://royalsocietypublishing.org/cms/asset/892d5993-0059-49c5-9606-123367e0b7d2/rsob200396f02.jpg)
Blood–brain barrier disruption in atrial fibrillation: a potential contributor to the increased risk of dementia and worsening of stroke outcomes? | Open Biology
![Neuroimmune Axes of the Blood–Brain Barriers and Blood–Brain Interfaces: Bases for Physiological Regulation, Disease States, and Pharmacological Interventions | Pharmacological Reviews Neuroimmune Axes of the Blood–Brain Barriers and Blood–Brain Interfaces: Bases for Physiological Regulation, Disease States, and Pharmacological Interventions | Pharmacological Reviews](https://pharmrev.aspetjournals.org/content/pharmrev/70/2/278/F6.large.jpg)
Neuroimmune Axes of the Blood–Brain Barriers and Blood–Brain Interfaces: Bases for Physiological Regulation, Disease States, and Pharmacological Interventions | Pharmacological Reviews
![Poldip2 mediates blood-brain barrier disruption in a model of sepsis-associated encephalopathy | Journal of Neuroinflammation | Full Text Poldip2 mediates blood-brain barrier disruption in a model of sepsis-associated encephalopathy | Journal of Neuroinflammation | Full Text](https://media.springernature.com/lw685/springer-static/image/art%3A10.1186%2Fs12974-019-1575-4/MediaObjects/12974_2019_1575_Fig8_HTML.png)
Poldip2 mediates blood-brain barrier disruption in a model of sepsis-associated encephalopathy | Journal of Neuroinflammation | Full Text
![In vitro modeling of blood–brain barrier and interface functions in neuroimmune communication | Fluids and Barriers of the CNS | Full Text In vitro modeling of blood–brain barrier and interface functions in neuroimmune communication | Fluids and Barriers of the CNS | Full Text](https://media.springernature.com/lw685/springer-static/image/art%3A10.1186%2Fs12987-020-00187-3/MediaObjects/12987_2020_187_Fig1_HTML.png)
In vitro modeling of blood–brain barrier and interface functions in neuroimmune communication | Fluids and Barriers of the CNS | Full Text
![Dual microglia effects on blood brain barrier permeability induced by systemic inflammation | Nature Communications Dual microglia effects on blood brain barrier permeability induced by systemic inflammation | Nature Communications](https://media.springernature.com/lw685/springer-static/image/art%3A10.1038%2Fs41467-019-13812-z/MediaObjects/41467_2019_13812_Fig7_HTML.png)
Dual microglia effects on blood brain barrier permeability induced by systemic inflammation | Nature Communications
![Lipopolysaccharide-induced blood-brain barrier disruption: roles of cyclooxygenase, oxidative stress, neuroinflammation, and elements of the neurovascular unit | Journal of Neuroinflammation | Full Text Lipopolysaccharide-induced blood-brain barrier disruption: roles of cyclooxygenase, oxidative stress, neuroinflammation, and elements of the neurovascular unit | Journal of Neuroinflammation | Full Text](https://media.springernature.com/lw685/springer-static/image/art%3A10.1186%2Fs12974-015-0434-1/MediaObjects/12974_2015_434_Fig6_HTML.gif)
Lipopolysaccharide-induced blood-brain barrier disruption: roles of cyclooxygenase, oxidative stress, neuroinflammation, and elements of the neurovascular unit | Journal of Neuroinflammation | Full Text
![Molecules | Free Full-Text | RhoA/ROCK-2 Pathway Inhibition and Tight Junction Protein Upregulation by Catalpol Suppresses Lipopolysaccaride-Induced Disruption of Blood-Brain Barrier Permeability | HTML Molecules | Free Full-Text | RhoA/ROCK-2 Pathway Inhibition and Tight Junction Protein Upregulation by Catalpol Suppresses Lipopolysaccaride-Induced Disruption of Blood-Brain Barrier Permeability | HTML](https://www.mdpi.com/molecules/molecules-23-02371/article_deploy/html/images/molecules-23-02371-g001.png)
Molecules | Free Full-Text | RhoA/ROCK-2 Pathway Inhibition and Tight Junction Protein Upregulation by Catalpol Suppresses Lipopolysaccaride-Induced Disruption of Blood-Brain Barrier Permeability | HTML
![Frontiers | The impact of microglial activation on blood-brain barrier in brain diseases | Cellular Neuroscience Frontiers | The impact of microglial activation on blood-brain barrier in brain diseases | Cellular Neuroscience](https://www.frontiersin.org/files/Articles/103334/fncel-08-00362-HTML/image_m/fncel-08-00362-g002.jpg)